环氧二十碳三烯酸通过线粒体途径保护缺血/再灌注损伤机制的研究进展
作者:
作者单位:

(1.广东医科大学附属医院麻醉科,广东湛江 524000; 2.广东省实验动物监测所,广东省实验动物重点实验室,广州 510633)

中图分类号:

R-33


Advances in understanding the mechanism by which epoxyeicosatrienoic acids protect against ischemia / reperfusion injury through the mitochondrial pathway
Author:
Affiliation:

(1. Department of Anesthesiology, Affiliated Hospital of Guangdong Medical University, Zhanjiang 524000, China.2. Guangdong Laboratory Animals Monitoring Institute; Guangdong Key Laboratory for Laboratory Animals,Guangzhou 510633)

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    摘要:

    越来越多的证据表明环氧二十碳三烯酸(epoxyeicosatrienoic acids,EETs)能够通过多种作用机制在组织器官缺血/ 再灌注(ischemia/ reperfusion,I/ R)损伤中发挥保护性作用,其中线粒体途径在这些作用机制中有着重要的地位?然而作为I/ R 损伤防治的重要靶点,线粒体途径在EETs 介导的I/ R 损伤保护机制中却未得到很深入的探讨?本文就线粒体K+通道?线粒体通透性转换孔?线粒体促凋亡蛋白以及线粒体结构完整性在EETs 减轻I/ R 损伤中所起的作用作一综述,为以后更深层次的研究提供理论基础,同时这对于寻找I/ R 新的防治方式和靶点?保护重要组织器官?改善患者的病情及预后都有着重要意义?

    Abstract:

    Increasing evidence suggests that epoxyeicosatrienoic acids (EETs) play a protective role in tissue and organ ischemia/ reperfusion ( I/ R) injury through a variety of mechanisms, and the mitochondrial pathway plays an important role in these mechanisms. However, as an important target for I/ R injury prevention and treatment, the mitochondrial pathway has not been explored in depth for EET-mediated protective mechanisms in I/ R injury. This review summarizes the role of mitochondrial K+ channels, mitochondrial permeability transition pores, mitochondrial proapoptotic proteins, and mitochondrial structural integrity in EET-mediated reductions of I/ R injury, providing a theoretical basis for further research. This is of great significance for preventing and treating I/ R injury of organs, and improving the prognosis of this disease.

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叶永才,陈哲璇,李想,谭伟江,杨丰华,张良清.环氧二十碳三烯酸通过线粒体途径保护缺血/再灌注损伤机制的研究进展[J].中国比较医学杂志,2019,29(11):105~110.

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  • 在线发布日期: 2019-12-10
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