SIVmac251感染增强恒河猴巨噬细胞对galectin-3诱导凋亡的敏感性
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国家自然科学基金项目(青年科学基金项目)


Enhancement of sensitivity to galectin-3-induced apoptosis of macrophage from SIVmac251-infected rhesus macaque
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The National Natural Science Foundation of China (Youth Program)

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    摘要:

    目的 研究SIVmac251感染对galectin-3诱导恒河猴巨噬细胞凋亡的影响。方法 从健康猴和SIVmac251感染猴外周血分离单核细胞,用GM-CSF与IL-6诱导单核细胞分化成巨噬细胞。巨噬细胞与galectin-3共孵育5.5小时后,细胞经PE-Annexin V/PerCP-7ADD染色,用流式细胞仪检测细胞凋亡情况。结果 无galectin-3诱导条件下健康猴与SIVmac251感染猴巨噬细胞中Annexin V阳性细胞的百分率分别为5.08%与4.87%。Galectin-3诱导条件下健康猴巨噬细胞中Annexin V阳性细胞的百分率升至17.62%, SIVmac251感染猴巨噬细胞中Annexin V阳性细胞的百分率升至45.56%,两者具有统计学差异(p﹤0.01)。结论 SIVmac251感染增强恒河猴巨噬细胞对galectin-3诱导凋亡的敏感性。

    Abstract:

    Objective To study the effect of SIVmac251-infection on apoptosis induction of macrophage from rhesus macaque by galectin-3. Methods Monocytes were isolated from peripheral blood of healthy or SIVmac251-infected rhesus macaques. Cells were induced to differentiate to macrophages by GM-CSF and IL-6. Macrophages were cultured for 5.5 hours at the presence of galectin-3. Cells were stained by PE-Annexin V/ PerCP-7ADD and apoptosis was examined upon flow cytometry. Results The percentages of Annexin V cells without galectin-3 induction were 5.08% and 4.87% for macrophages from healthy or SIVmac251-infected rhesus macaques respectively. The percentages of them with galectin-3 induction increased to 17.62% and 45.56% respectively. The increase for SIVmac251-infected rhesus macaque was significantly greater than that for healthy monkey (p﹤0.01). Conclusion SIVmac251-infection results in sensitivity enhancement to apoptosis induction of macrophage from rhesus macaque by galectin-3.

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薛婧,魏强. SIVmac251感染增强恒河猴巨噬细胞对galectin-3诱导凋亡的敏感性[J].中国比较医学杂志,2013,23(10).

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  • 收稿日期:2013-09-04
  • 最后修改日期:2013-09-06
  • 录用日期:2013-10-14
  • 在线发布日期: 2013-12-16
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